A recent study indicates that tau protein and beta-amyloid can protect against neuron damage from herpes (HSV-1) infections, but prolonged accumulation may lead to toxicity and contribute to Alzheimer's. Despite substantial funding and research over decades, effective Alzheimer's treatments remain elusive. Recent data links HSV-1 infection to increased Alzheimer's risk, prompting a pharmaceutical team's call to prioritize herpes prevention as a public health initiative. Studies also suggest a correlation between shingles vaccination and reduced Alzheimer's risk, though more research is needed to establish direct causation.
"A recent study shows that the tau protein protects against neuronal damage caused by herpes (HSV-1) infection, but over time it can become toxic."
"Preventing herpesvirus infections should be considered a public health priority as it may reduce the risk of dementia, based on a large-scale health database analysis."
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