"Researchers have identified a network of neurons that could explain why stress fuels the symptoms of eczema. In mice with atopic dermatitis, these neurons produce inflammatory proteins in response to stress signals from the central nervous system."
"These molecules lure immune cells called eosinophils to the skin, which drive further inflammation. The findings could lead to targeted treatments that block the activity of these nerves, or the molecules they produce, if the same mechanism is at play in people."
Researchers have identified a network of stress-responsive skin neurons that produce inflammatory proteins, worsening eczema symptoms. In mice with atopic dermatitis, these neurons respond to stress signals from the central nervous system, attracting immune cells called eosinophils to the skin and driving inflammation. This discovery may pave the way for targeted treatments that inhibit the activity of these nerves or the inflammatory molecules they produce, assuming similar mechanisms exist in humans.
Read at Nature
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